酰基辅酶A合成酶4重组兔单抗
Rrmab®兔单抗
货号：bsm-62432R
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概述
产品编号
bsm-62432R
产品类型
重组兔单抗、mIHC精品抗体
英文名称
ACSL4 Recombinant Rabbit mAb
中文名称
酰基辅酶A合成酶4重组兔单抗
英文别名
ACS4; FACL4; LACS4; MRX63; MRX68; XLID63; 9430020A05Rik; ACSL4_HUMAN; ACSL4; Arachidonate--CoA ligase; Long-chain acyl-CoA synthetase 4 (LACS 4); 6.2.1.3; ACSL4_MOUSE; Long-chain acyl-CoA synthetase 4 (LACS 4 | mACS4); ACSL4_RAT;
抗体来源
Rabbit
免疫原
A synthesized peptide derived from human ACSL4: 650-691
亚型
IgG
性状
Liquid
纯化方法
affinity purified by Protein A
克隆类型
Recombinant
克隆号
1G12
理论分子量
79
检测分子量
79
储存液
10mM phosphate buffered saline(pH 7.4) with 150mM sodium chloride, 0.05% BSA, 0.02% Proclin300 and 50% glycerol.
研究领域
SWISS
Gene ID
保存条件
Store at 4℃ for short term. Store at -20℃ for long term. Avoid repeated freeze/thaw cycles.
注意事项
This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications.
数据库链接
背景资料
Catalyzes the conversion of long-chain fatty acids to their active form acyl-CoA for both synthesis of cellular lipids, and degradation via beta-oxidation. Preferentially activates arachidonate and eicosapentaenoate as substrates.
产品应用
| 应用 | 已检合格种属 | 预测种属 | 推荐稀释比例 |
|---|---|---|---|
| WB | Human, Mouse | Rat | 1:500-2000 |
| IHC-P | Human, Mouse, Rat | 1:100-500 | |
| IHC-F | Human, Mouse, Rat | 1:100-500 | |
| IF | Human, Mouse, Rat | 1:100-500 | |
| IP | Human, Mouse, Rat | 1:20-50 |
交叉反应
交叉反应: Human, Mouse, Rat
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靶标
基因名
ACSL4
蛋白名
Long-chain-fatty-acid--CoA ligase 4
亚细胞定位
Mitochondrion outer membrane. Peroxisome membrane. Microsome membrane. Endoplasmic reticulum membrane.
疾病
Defects in ACSL4 are the cause of mental retardation X-linked type 63 (MRX63) [MIM:300387]. Mental retardation is a mental disorder characterized by significantly sub-average general intellectual functioning associated with impairments in adaptative behavior and manifested during the developmental period. Non-syndromic mental retardation patients do not manifest other clinical signs.
Defects in ACSL4 are involved in Alport syndrome with mental retardation midface hypoplasia and elliptocytosis (ATS-MR) [MIM:300194]. A X-linked contiguous gene deletion syndrome characterized by glomerulonephritis, deafness, mental retardation, midface hypoplasia and elliptocytosis.
Defects in ACSL4 are involved in Alport syndrome with mental retardation midface hypoplasia and elliptocytosis (ATS-MR) [MIM:300194]. A X-linked contiguous gene deletion syndrome characterized by glomerulonephritis, deafness, mental retardation, midface hypoplasia and elliptocytosis.
相似性
Belongs to the ATP-dependent AMP-binding enzyme family.
功能
Activation of long-chain fatty acids for both synthesis of cellular lipids, and degradation via beta-oxidation. Preferentially uses arachidonate and eicosapentaenoate as substrates.
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具体参考文献:bsm-62432R 被引用于3文献中
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